Patented innovation behind the products - from cellular activation to skin care and product synergy.
Backed by Patents. Built on Product Innovation.
LifeVantage has developed a portfolio of patented and patent-published technologies connected to cellular activation, oxidative stress pathways, skin care, collagen support and product synergy.
Patents are one way to see where a company has invested in original formulation, product-combination science and long-term innovation.
LifeVantage is known for creating products designed to work with the body’s own biological pathways. From the original Protandim Nrf2 Synergizer to newer product combinations like the Healthy Glow Essentials Stack, the company’s patent history helps tell the story of a brand focused on research, formulation and activation-based wellness.
Below you’ll find LifeVantage patent records organised by product family.
The Product That Started It All
Protandim NRF2 Synergizer Patents
Protandim Nrf2 Synergizer is the product that helped establish LifeVantage’s activation-based approach.
The original Protandim Nrf2 patent family included U.S. and international patent records connected to compositions and methods for alleviating oxidative stress, supporting antioxidant enzyme activity and related biological pathways.
These patents are included here as part of the product’s innovation history. LifeVantage’s public filings state that the original U.S. patents protecting Protandim Nrf2 expired in March 2025, so they should be described as historical patent records rather than current active protection.
Independent Studies
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Protandim vs individual ingredients for HO-1 activation
Cell Study
University of Colorado / Denver VA Medical Center
This study looked at the components of Protandim and their ability to induce heme oxygenase-1, also known as HO-1. The important part is that the study explored the synergy of the full ingredient combination, rather than only looking at each ingredient in isolation.
Simple takeaway:
The study found that the full Protandim combination worked better than the ingredients on their own for inducing HO-1, an important antioxidant and stress-response enzyme. It also reported increases in total glutathione in Protandim-treated cells, supporting the idea that the formula works through synergy rather than one “hero” ingredient.
Protandim vs oxidative stress in human coronary artery cells
Cell Study
Colorado State University
This study investigated whether Protandim could activate Nrf2 in human coronary artery endothelial cells and protect those cells against an oxidative challenge. The researchers focused on Nrf2 nuclear localisation and phase II antioxidant enzyme expression.
Simple takeaway:
The study found that Protandim induced Nrf2 nuclear localisation, increased antioxidant enzyme expression and helped protect human coronary artery endothelial cells from oxidative challenge. In simple terms, it showed Protandim activating the cellular antioxidant-response pathway in a human vascular cell model.
Protandim vs intimal hyperplasia in human saphenous veins
Ex Vivo Human Tissue Study
The Ohio State University
This study looked at human saphenous veins cultured outside the body and explored whether Protandim could attenuate intimal hyperplasia through a catalase-dependent pathway.
Simple takeaway:
The study found that Protandim reduced intimal hyperplasia, abnormal thickening inside the vein wall, in cultured human saphenous veins. The effect appeared to depend on catalase, which links the result back to Protandim’s role in supporting antioxidant enzyme activity.
Nrf2 activation vs oxidative stress in health and disease
Review + Pathway Research
University of Colorado Denver
This review explored oxidative stress, Nrf2 activation and the broader therapeutic potential of activating the body’s antioxidant-response pathways. The paper also discusses gene-expression data from cultured human cells treated with Protandim, including pathway analysis connected to antioxidant and stress-response systems.
Simple takeaway:
This review positioned Nrf2 as a major pathway in the body’s response to oxidative stress. It also discussed Protandim-related pathway analysis, showing that Nrf2 activation influenced genes and pathways connected to antioxidant enzymes, cellular stress response and disease-related biological processes.
Protandim vs two-stage skin carcinogenesis in mice
Animal / Model Research
Louisiana State University Health Sciences Center
This PLOS ONE study tested Protandim in a two-stage mouse skin carcinogenesis model. The study reported that mice fed a Protandim-containing diet had reduced tumour incidence and multiplicity compared with the basal diet group, while also examining oxidative stress, antioxidant enzyme activity, inflammation and cell-proliferation markers.
Simple takeaway:
The study found that mice receiving Protandim had 33% lower tumour incidence and 57% lower tumour multiplicity compared with mice on the basal diet. It also found increased SOD and catalase activity, along with reduced markers of oxidative stress, inflammation and cell proliferation in the model.
Protandim vs lifespan in genetically diverse mice
Animal / Model Research
National Institute on Aging
This National Institute on Aging Interventions Testing Program paper tested several agents in genetically heterogeneous mice, including Protandim. The study reported that Protandim, described as an Nrf2 inducer, was associated with longer lifespan in male mice.
Simple takeaway:
The study found that Protandim extended median lifespan in male mice only. It did not show the same effect in female mice, which makes the finding interesting but also important to interpret carefully. The value of this study is that Protandim was tested inside a recognised ageing-research program using genetically diverse mice.
Protandim vs oxidative insult in oligodendrocytes
Cell Study
VU University Medical Center
This study explored Protandim in oligodendrocytes under oxidative insult. The research reported that Protandim induced antioxidant activity in oligodendrocytes and helped protect the cells against oxidative insult.
Simple takeaway:
The study found that Protandim helped prevent ROS-driven oligodendrocyte cell death and promoted oligodendrocyte precursor cell differentiation under oxidative stress conditions. In simple terms, it showed Protandim supporting cell survival and antioxidant defence in a nervous-system cell model.
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Journals
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Protandim vs individual ingredients for HO-1 activation
Cell Study
University of Colorado / Denver VA Medical Center
This study looked at the components of Protandim and their ability to induce heme oxygenase-1, also known as HO-1. The important part is that the study explored the synergy of the full ingredient combination, rather than only looking at each ingredient in isolation.
Simple takeaway:
The study found that the full Protandim combination worked better than the ingredients on their own for inducing HO-1, an important antioxidant and stress-response enzyme. It also reported increases in total glutathione in Protandim-treated cells, supporting the idea that the formula works through synergy rather than one “hero” ingredient.
Protandim vs oxidative stress in human coronary artery cells
Cell Study
Colorado State University
This study investigated whether Protandim could activate Nrf2 in human coronary artery endothelial cells and protect those cells against an oxidative challenge. The researchers focused on Nrf2 nuclear localisation and phase II antioxidant enzyme expression.
Simple takeaway:
The study found that Protandim induced Nrf2 nuclear localisation, increased antioxidant enzyme expression and helped protect human coronary artery endothelial cells from oxidative challenge. In simple terms, it showed Protandim activating the cellular antioxidant-response pathway in a human vascular cell model.
Protandim vs intimal hyperplasia in human saphenous veins
Ex Vivo Human Tissue Study
The Ohio State University
This study looked at human saphenous veins cultured outside the body and explored whether Protandim could attenuate intimal hyperplasia through a catalase-dependent pathway.
Simple takeaway:
The study found that Protandim reduced intimal hyperplasia, abnormal thickening inside the vein wall, in cultured human saphenous veins. The effect appeared to depend on catalase, which links the result back to Protandim’s role in supporting antioxidant enzyme activity.
Nrf2 activation vs oxidative stress in health and disease
Review + Pathway Research
University of Colorado Denver
This review explored oxidative stress, Nrf2 activation and the broader therapeutic potential of activating the body’s antioxidant-response pathways. The paper also discusses gene-expression data from cultured human cells treated with Protandim, including pathway analysis connected to antioxidant and stress-response systems.
Simple takeaway:
This review positioned Nrf2 as a major pathway in the body’s response to oxidative stress. It also discussed Protandim-related pathway analysis, showing that Nrf2 activation influenced genes and pathways connected to antioxidant enzymes, cellular stress response and disease-related biological processes.
Protandim vs two-stage skin carcinogenesis in mice
Animal / Model Research
Louisiana State University Health Sciences Center
This PLOS ONE study tested Protandim in a two-stage mouse skin carcinogenesis model. The study reported that mice fed a Protandim-containing diet had reduced tumour incidence and multiplicity compared with the basal diet group, while also examining oxidative stress, antioxidant enzyme activity, inflammation and cell-proliferation markers.
Simple takeaway:
The study found that mice receiving Protandim had 33% lower tumour incidence and 57% lower tumour multiplicity compared with mice on the basal diet. It also found increased SOD and catalase activity, along with reduced markers of oxidative stress, inflammation and cell proliferation in the model.
Protandim vs lifespan in genetically diverse mice
Animal / Model Research
National Institute on Aging
This National Institute on Aging Interventions Testing Program paper tested several agents in genetically heterogeneous mice, including Protandim. The study reported that Protandim, described as an Nrf2 inducer, was associated with longer lifespan in male mice.
Simple takeaway:
The study found that Protandim extended median lifespan in male mice only. It did not show the same effect in female mice, which makes the finding interesting but also important to interpret carefully. The value of this study is that Protandim was tested inside a recognised ageing-research program using genetically diverse mice.
Protandim vs oxidative insult in oligodendrocytes
Cell Study
VU University Medical Center
This study explored Protandim in oligodendrocytes under oxidative insult. The research reported that Protandim induced antioxidant activity in oligodendrocytes and helped protect the cells against oxidative insult.
Simple takeaway:
The study found that Protandim helped prevent ROS-driven oligodendrocyte cell death and promoted oligodendrocyte precursor cell differentiation under oxidative stress conditions. In simple terms, it showed Protandim supporting cell survival and antioxidant defence in a nervous-system cell model.
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
U.S. Patent No. 9,265,808 B2
Compositions for alleviating inflammation and oxidative stress in a mammal
U.S. Patent No. 8,435,574 B2
Compositions for alleviating inflammation and oxidative stress in a mammal
U.S. Patent No. 8,221,805 B2
Compositions for alleviating inflammation and oxidative stress in a mammal
U.S. Patent No. 7,923,045 B2
Compositions for alleviating inflammation and oxidative stress in a mammal
U.S. Patent No. 7,579,026 B2
Methods for enhancing antioxidant enzyme activity and reducing c-reactive protein levels
U.S. Patent No. 7,384,655 B2
Preparation of compositions to alleviate inflammation and oxidative stress in a mammal
International Patent
Australian Patent AU2005229008B2
Compositions and method for alleviating inflammation and oxidative stress in a mammal
International Patent
Canadian Patent CA2560826C
Antioxidant-promoting herbal extract compositions for alleviating oxidative stress in a mammal
International Patent
Chinese Patent CN1956726B / ZL200580016510.7
Compositions and method for alleviating inflammation and oxidative stress in a mammal
International Patent
Japanese Patent JP5507645B2
Compositions and methods for reducing inflammation and oxidative stress in mammals
International Patent
European Patent EP1740196B1
Compositions and method for alleviating inflammation and oxidative stress in a mammal
International Patent
Indian Patent No. 248562
An antioxidant-promoting composition
Independent Studies
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Protandim vs individual ingredients for HO-1 activation
Cell Study
University of Colorado / Denver VA Medical Center
This study looked at the components of Protandim and their ability to induce heme oxygenase-1, also known as HO-1. The important part is that the study explored the synergy of the full ingredient combination, rather than only looking at each ingredient in isolation.
Simple takeaway:
The study found that the full Protandim combination worked better than the ingredients on their own for inducing HO-1, an important antioxidant and stress-response enzyme. It also reported increases in total glutathione in Protandim-treated cells, supporting the idea that the formula works through synergy rather than one “hero” ingredient.
Protandim vs oxidative stress in human coronary artery cells
Cell Study
Colorado State University
This study investigated whether Protandim could activate Nrf2 in human coronary artery endothelial cells and protect those cells against an oxidative challenge. The researchers focused on Nrf2 nuclear localisation and phase II antioxidant enzyme expression.
Simple takeaway:
The study found that Protandim induced Nrf2 nuclear localisation, increased antioxidant enzyme expression and helped protect human coronary artery endothelial cells from oxidative challenge. In simple terms, it showed Protandim activating the cellular antioxidant-response pathway in a human vascular cell model.
Protandim vs intimal hyperplasia in human saphenous veins
Ex Vivo Human Tissue Study
The Ohio State University
This study looked at human saphenous veins cultured outside the body and explored whether Protandim could attenuate intimal hyperplasia through a catalase-dependent pathway.
Simple takeaway:
The study found that Protandim reduced intimal hyperplasia, abnormal thickening inside the vein wall, in cultured human saphenous veins. The effect appeared to depend on catalase, which links the result back to Protandim’s role in supporting antioxidant enzyme activity.
Nrf2 activation vs oxidative stress in health and disease
Review + Pathway Research
University of Colorado Denver
This review explored oxidative stress, Nrf2 activation and the broader therapeutic potential of activating the body’s antioxidant-response pathways. The paper also discusses gene-expression data from cultured human cells treated with Protandim, including pathway analysis connected to antioxidant and stress-response systems.
Simple takeaway:
This review positioned Nrf2 as a major pathway in the body’s response to oxidative stress. It also discussed Protandim-related pathway analysis, showing that Nrf2 activation influenced genes and pathways connected to antioxidant enzymes, cellular stress response and disease-related biological processes.
Protandim vs two-stage skin carcinogenesis in mice
Animal / Model Research
Louisiana State University Health Sciences Center
This PLOS ONE study tested Protandim in a two-stage mouse skin carcinogenesis model. The study reported that mice fed a Protandim-containing diet had reduced tumour incidence and multiplicity compared with the basal diet group, while also examining oxidative stress, antioxidant enzyme activity, inflammation and cell-proliferation markers.
Simple takeaway:
The study found that mice receiving Protandim had 33% lower tumour incidence and 57% lower tumour multiplicity compared with mice on the basal diet. It also found increased SOD and catalase activity, along with reduced markers of oxidative stress, inflammation and cell proliferation in the model.
Protandim vs lifespan in genetically diverse mice
Animal / Model Research
National Institute on Aging
This National Institute on Aging Interventions Testing Program paper tested several agents in genetically heterogeneous mice, including Protandim. The study reported that Protandim, described as an Nrf2 inducer, was associated with longer lifespan in male mice.
Simple takeaway:
The study found that Protandim extended median lifespan in male mice only. It did not show the same effect in female mice, which makes the finding interesting but also important to interpret carefully. The value of this study is that Protandim was tested inside a recognised ageing-research program using genetically diverse mice.
Protandim vs oxidative insult in oligodendrocytes
Cell Study
VU University Medical Center
This study explored Protandim in oligodendrocytes under oxidative insult. The research reported that Protandim induced antioxidant activity in oligodendrocytes and helped protect the cells against oxidative insult.
Simple takeaway:
The study found that Protandim helped prevent ROS-driven oligodendrocyte cell death and promoted oligodendrocyte precursor cell differentiation under oxidative stress conditions. In simple terms, it showed Protandim supporting cell survival and antioxidant defence in a nervous-system cell model.
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Journals
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Protandim vs individual ingredients for HO-1 activation
Cell Study
University of Colorado / Denver VA Medical Center
This study looked at the components of Protandim and their ability to induce heme oxygenase-1, also known as HO-1. The important part is that the study explored the synergy of the full ingredient combination, rather than only looking at each ingredient in isolation.
Simple takeaway:
The study found that the full Protandim combination worked better than the ingredients on their own for inducing HO-1, an important antioxidant and stress-response enzyme. It also reported increases in total glutathione in Protandim-treated cells, supporting the idea that the formula works through synergy rather than one “hero” ingredient.
Protandim vs oxidative stress in human coronary artery cells
Cell Study
Colorado State University
This study investigated whether Protandim could activate Nrf2 in human coronary artery endothelial cells and protect those cells against an oxidative challenge. The researchers focused on Nrf2 nuclear localisation and phase II antioxidant enzyme expression.
Simple takeaway:
The study found that Protandim induced Nrf2 nuclear localisation, increased antioxidant enzyme expression and helped protect human coronary artery endothelial cells from oxidative challenge. In simple terms, it showed Protandim activating the cellular antioxidant-response pathway in a human vascular cell model.
Protandim vs intimal hyperplasia in human saphenous veins
Ex Vivo Human Tissue Study
The Ohio State University
This study looked at human saphenous veins cultured outside the body and explored whether Protandim could attenuate intimal hyperplasia through a catalase-dependent pathway.
Simple takeaway:
The study found that Protandim reduced intimal hyperplasia, abnormal thickening inside the vein wall, in cultured human saphenous veins. The effect appeared to depend on catalase, which links the result back to Protandim’s role in supporting antioxidant enzyme activity.
Nrf2 activation vs oxidative stress in health and disease
Review + Pathway Research
University of Colorado Denver
This review explored oxidative stress, Nrf2 activation and the broader therapeutic potential of activating the body’s antioxidant-response pathways. The paper also discusses gene-expression data from cultured human cells treated with Protandim, including pathway analysis connected to antioxidant and stress-response systems.
Simple takeaway:
This review positioned Nrf2 as a major pathway in the body’s response to oxidative stress. It also discussed Protandim-related pathway analysis, showing that Nrf2 activation influenced genes and pathways connected to antioxidant enzymes, cellular stress response and disease-related biological processes.
Protandim vs two-stage skin carcinogenesis in mice
Animal / Model Research
Louisiana State University Health Sciences Center
This PLOS ONE study tested Protandim in a two-stage mouse skin carcinogenesis model. The study reported that mice fed a Protandim-containing diet had reduced tumour incidence and multiplicity compared with the basal diet group, while also examining oxidative stress, antioxidant enzyme activity, inflammation and cell-proliferation markers.
Simple takeaway:
The study found that mice receiving Protandim had 33% lower tumour incidence and 57% lower tumour multiplicity compared with mice on the basal diet. It also found increased SOD and catalase activity, along with reduced markers of oxidative stress, inflammation and cell proliferation in the model.
Protandim vs lifespan in genetically diverse mice
Animal / Model Research
National Institute on Aging
This National Institute on Aging Interventions Testing Program paper tested several agents in genetically heterogeneous mice, including Protandim. The study reported that Protandim, described as an Nrf2 inducer, was associated with longer lifespan in male mice.
Simple takeaway:
The study found that Protandim extended median lifespan in male mice only. It did not show the same effect in female mice, which makes the finding interesting but also important to interpret carefully. The value of this study is that Protandim was tested inside a recognised ageing-research program using genetically diverse mice.
Protandim vs oxidative insult in oligodendrocytes
Cell Study
VU University Medical Center
This study explored Protandim in oligodendrocytes under oxidative insult. The research reported that Protandim induced antioxidant activity in oligodendrocytes and helped protect the cells against oxidative insult.
Simple takeaway:
The study found that Protandim helped prevent ROS-driven oligodendrocyte cell death and promoted oligodendrocyte precursor cell differentiation under oxidative stress conditions. In simple terms, it showed Protandim supporting cell survival and antioxidant defence in a nervous-system cell model.
Protandim vs oxidative stress in healthy adults
Human Study
University of Colorado Denver Health Sciences Center / Webb-Waring Institute for Cancer
In this published human study, Protandim was given to healthy adults aged 20 to 78. Researchers reported that after 30 days, TBARS, a marker used to measure oxidative stress, declined by an average of 40%. By 120 days, erythrocyte SOD activity increased by 30% and catalase activity increased by 54%.
Simple takeaway:
This study found that the finished Protandim formula significantly reduced an oxidative stress marker in healthy adults, while also increasing two of the body’s own antioxidant enzymes: SOD and catalase. In simple terms, it showed Protandim was not acting like a standard antioxidant supplement, it was associated with increased internal antioxidant enzyme activity.
Protandim Nrf2 Synergizer® + TrueScience® Liquid Collagen
Healthy Glow Essentials Stack®
The Healthy Glow Essentials Stack brings together two of LifeVantage’s best-known product technologies: Protandim Nrf2 Synergizer and TrueScience Liquid Collagen.
In 2026, LifeVantage announced a newly issued U.S. patent connected to this product combination. The patent relates to nutritional supplement compositions and methods for activating cellular signalling genes in innate inflammatory pathways.
This is a strong example of LifeVantage continuing to build beyond individual products and into product systems designed to work together.
U.S. Patent No. 12,589,067 B2
Composition and methods for activating cellular signaling genes in innate inflammatory pathways
Nrf2 + NRF1 + NAD Pathway Support
Protandim Tri-Synergizer®
Protandim Tri-Synergizer was created around three important activation pathways: Nrf2, NRF1 and NAD.
Rather than focusing on only one pathway, this patented product combination reflects LifeVantage’s broader approach to cellular health and product synergy. The patent covers compositions and methods for activating cellular signalling pathways.
U.S. Patent No. 11,484,563 B2
Compositions and methods for activating cellular signaling pathways
Topical Skin Care Technology
TrueScience® Skin Care
TrueScience Skin Care was developed around topical compositions connected to oxidative stress response in the skin.
These patent records show LifeVantage’s innovation extending beyond nutritional supplements and into personal care products designed to support the appearance and condition of the skin.
U.S. Patent No. 9,289,374 B2
Topical compositions and methods for reducing oxidative stress
Patent-Pending Innovation
MindBody System®
The MindBody GLP-1 System, also known in some international markets as the MB System, is LifeVantage’s patent-pending dual-product system made up of MB Core™ and MB Enhance™.
This product system reflects LifeVantage’s continued focus on activation-based wellness, combining ingredients designed to support the body’s natural GLP-1 pathway and gut-brain axis.
LifeVantage has filed a U.S. patent application for the MindBody GLP-1 System. If granted, the patent would protect the combined effects of the system.
Innovation You Can Trace.
In a crowded wellness market, many companies rely on ingredient trends, marketing language or short-term product hype.
LifeVantage has a different story.
Its patent history shows a long-term commitment to product formulation, cellular signalling pathways, topical skin care innovation and product-combination science.
For customers, this provides another layer of confidence. For people exploring the business, it shows a company continuing to invest in products, intellectual property and long-term innovation.
Start with the product that started it all.
If you are new to LifeVantage, the best place to begin is with the product that introduced many people to activation science:
Protandim NRF2 Synergizer.
The little yellow pill. The product at the centre of the LifeVantage research story. The place where the AgeWell Movement begins.
Speak to the person who shared this site with you.
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
Guy Hawkins
President of Sales
